Doctor Tullio Simoncini

THE WHOLE THERAPY

If 40, 30, or even 20 years ago it was still possible to somehow convince people of the goodness of official oncology and of its results, today, after results that are as continuous as they are inane, although trumpeted regularly by the media, nobody accepts being seduced by words, about hypotheses and promises that are undelivered and undeliverable, any longer. The painful awareness, which almost everyone has experienced, of the miserable end of this or that relative, friend, or acquaintance, is associated with these failures.
We must surrender to the evidence that contemporary oncology is incapable of giving us the answers and the necessary to those who are cancer patients and that therefore it is our moral and ethical obligation to try to find the correct solution for the gravest and most painful disease of our time.

Cancer is a fungus
For about 100 years, the fundamental theory behind cancer has been based on the hypothesis that it is a malfunctioning of the genes. This point of view implies that cancer is intracellular. My point of view however is that cancer is a fungal infection, and therefore an extra cellular phenomenon.

Candida
In the plant world, cancers are caused by a fungal invasion, and it is possible to argue that the same thing happens in human beings. Fungi are always involved in cancers: they are found both in vivo and in the post-mortem examination*.
However, scientists believe that they develop after the onset of the illness. My opinion is that they come before it: they produce the cancer, blunt the immune system and then invade the entire organism.
Each type of cancer is caused by fungi of the Candida species, as also referenced by other research*, and the histological configuration is a result of the defence reaction of a tissue against the invasion. In time, the tissue gets exhausted and produces only undifferentiated cells.
A cancer could be termed a ‘solid abscess,’ where the colonies form the centre, and host cellular reaction is all around.

Sodium bicarbonate
Traditional anti-fungal drugs are ineffective in treating tumours because the solid colonies can be attacked only on the surface of their volume, and after the first administrations they become resistant.
A solid infection is much more powerful than a bacterial one. That is why simple fungal infections can last forever.
I have identified the substances uniquely able to penetrate these volumetric infections: for cancer of the internal organs it is sodium bicarbonate; and the best substance to eliminate skin cancer is iodine tincture, particularly when it is spread onto the growth. There are many publications that describe the effectiveness of sodium bicarbonate on cancer*, but the conclusions drawn in them are invariably wrong because they assume intracellular, rather than antifungal action.

The treatment
My methods have cured people for 20 years. Many of my patients recovered completely from cancer, even in cases where official oncology had given up.
The best way to try to eliminate a tumour is to bring it into contact with sodium bicarbonate, as closely as possible, i.e. using oral administration for the digestive tract, enemas for the rectum, douching for the vagina and uterus, intravenous injection for the lung and the brain, and inhalation for the upper airways. Breasts, lymph nodes and subcutaneous lumps can be treated with local perfusions. The internal organs can be treated with sodium bicarbonate by locating suitable catheters in the arteries (of the liver, pancreas, prostate, and limbs) or in the cavities (of the pleura or peritoneum). It is important to treat each type of cancer with the right dosage. For a phleboclysis, 500 cc at 5% or 8,4% is required; for external administrations it is enough to taste if the solution is salty. Sometimes it is judicious to combine different administrations. For each treatment, take into consideration that tumour colonies regress between the third and fourth day and collapse between the fourth and fifth, so that a six day administration is sufficient.
A complete, effective cycle is made up of six treatment days on, and six days off, repeated four times. The most important side effects of this care system are thirst and weakness.
For skin cancer, a 7% iodine tincture should be spread on the affected area, 20-30 times once a day, with the aim of producing a number of layers of crusts. After this treatment, the cancer will be gone and stay away forever.

Pediatric oncology
This therapy is also applicable in paediatric oncology, provided the dosage is adjusted and revised according to the weight and age of the infant, as well as the type of neoplastic formation.

Clinical cases
Several well-documented clinical cases, examined before and after sodium bicarbonate treatments, were reported on and outlined on www.cancerfungus.com.
I wrote a book entitled ‘Cancer is a Fungus’ published in Italian, Dutch and English, thanks to the help of family and friends. Video footage of patient testimonials is also available on the internet site mentioned above. My deep wish is to make this therapy available to all humanity. In order to do so I must carry on my research. This research is done privately, since I have received no response from official institutions — despite having published the results of the well-documented and successful clinical cases I have treated. It is my firm hope that soon, the fundamental role of fungi in the development of neoplastic disease is acknowledged, so that it is possible to find, with the help of all existing forces of the health establishment, those anti-mycotic drugs and those systems of therapy that can quickly defeat, without damage and suffering, a disease that brings so much devastation to humanity.

When compared with the whole universe of fungi forms mycetes that are pathogenic for humans are not very numerous.

They usually cause diseases called mycosis, which are commonly divided into superficial (when the infection is limited to the cutis, body hair, hair of the head, and nails) and deep (when the infection attacks internal organs such as lung, intestine encephalus, bones, and others). The fungi are generally classified as:

1. dermatorphytes, causing afflictions that are typical of the epidermis (tinea)
2. sporotrichum schenckii, which are also almost exclusively located on the epidermis
3. Criptococcus neoformans, responsible for a diffused infection of the lung (the organisms are inhaled with dust) as well as chronic meningitis
4. Histoplasma capsulatum, which in humans produces the nodular cutaneous form, mucous form, the pulmonary form, and the systemic form.
5. Actinomycetes, with pathogenic action on the cutis, lungs, and intestine
6. Chrysosporium parvum (causal agent of the adiasphyromycosis), a cosmopolitan disease where the respiratory tract constitutes the primary and only localization of the infection
7. Aspergillus fumigatus, cause of the Aspergillosis, whose most frequent location is in the lungs, followed by a secondary location in the cerebrum and in the kidneys
8. Paracocci dioides brasiliensis, which causes the paracoccidioidomycosis, a primary pulmonary infection that can become diffused in immuno-depressed patients
9. In recent years, Pheoiphomycosis ialiphmycosis, pennicilinosis (marneffei), zigomicosis and other rare mycotic infections are acquiring more and more importance since they can be responsible for pathological scenarios that are sometimes very serious because of the compromised conditions of immuno-compromised patients.
10. Candida, both as Albicans and as any other pathogenic stock which afflicts the cutis, nails, internal mucus membranes (oral cavity, vulvar vaginitis, urethritis, balanitis, perianal infection), bronchi and lungs.
Candida is also responsible for causing generalized forms of septicemia of remarkable gravity.

The gravest disease of humanity is, therefore, hidden within this grouping of fungi. Some further analysis will make it easier to identify the cause.

Dermatorphytes and sporotrichum are responsible for a morbidity that is too specific. We know from experience that Actinomycetes, Criptococcus, Hystoplasm, Chrysosporium, Paracoccidioides and other causal agents of Pheoiphomycosis ialiphmycosis, pennicilinosis, zigomicosis are very rarely part of a pathological context. Finally, Aspergillus can be considered a variation of Candida. Only one of the six kinds described above remains as the sole responsible agent for tumors: Candida.

CANDIDA AND CANCER ALWAYS CONCURRENT

There are a large number of works that document the constant presence of the mycetes in the tissues of cancer patients, especially in terminal patients.

In recent years, we have observed a crescendo of voices addressing this terrible fungus to the point of defining it as 'the most important and most urgent problem that oncology has to solve'.

The following figures concerning the coexistence of Candida and cancer have been collected by several authors:[53]

R.L. Hopfer: 79%
U. Kaben: 80%
W. T. Hughes: 91 %
T.E. Kiehn: 97%
The percentages observed are truly impressive, especially when considering the difficulty of seeing Candida in the organic materials to be examined. This was also reported by R.S. Escuro, Z. O. Karaev, and T.J. Walsh.[54]

The positive results quoted allow us to confirm that Candida is always present in the tissues of cancer patients. Not only that, but Candida species represent today, according to several scholars, the first cause of morbidity and mortality in patients affected by neoplasias of the hemolinphopoietic system.[55]

O. Uzun even analyzed all data from 1974 to 1999 concerning the presence of candidosis in patients and the prognostic factors including predictable elements of mortality and came to the conclusion that the global rate of mortality in cancer patients varies between 33% and 75% and that this is independent of the type of infecting Candida.[56]

The phenomenon is usually interpreted as a consequence of the weakening and of the exhaustion of the organism because of neoplastic lesions. Conversely, we have to believe that the aggression of Candida takes place in the carcinogenic sense after the superficial pathogenic phases - that is, the classic epithelial candidosis - in several stages:

a) rooting in the deep connective tissue (in the various organs)

b) expansion with evoking of an organic reaction that attempts to encyst the fungin colonies, with the outcome being the formation of neoplasias

c) growth both in the surrounding tissue and remotely (metastasis).

d) progressive exhaustion of the organism with consequential global organism invasion. This is the stage that is most commonly observed and that is considered 'opportunistic'

e) exitus

In summary, Candida is not a post hoc but an ante hoc cause.

Tumors are perceived as one phenomenon.

Tumors are one phenomenon, but there are many types. Why?

According to official views that see genetic alteration at the basis of neoplastic development, it is possible that the alteration can manifest itself in any environment with all possible typological differentiations.
From the microbiological point of view, instead, it is always Candida that invades various anatomical parts, evoking different reactions as a function of the organs it feeds on. These behaviors are a function of the quantity and quality of the affected tissues. An organ whose connective tissue has been invaded defends itself with cellular hyper-productions that attempt to encyst the fungin colonies which are trying to completely colonize the organism.

It is in this way that the whole histological variety of neoplasias can be explained. The histological variety appears not to be influential in the determination of the cause, which is always and only Candida.
It is in this way that during a neoplastic event some genes can be hyper-expressed - that is, amplified - in a defensive effort determined by hyper-productive needs of the tissue. This reaction is normal and not anomalous at all.

Consider the following example. If we take an inert thorn, for example that of a sea urchin, and we inoculate it first in the skin, then in the bronchi, the bone, brain and in other body areas, we evoke an immune response of a cellular type tending to encyst the thorn, that is, to form some kind of a cocoon in which to enclose it.

By the same token, the immune system interprets fungin colonies beyond a certain dimension as extraneous foreign bodies stimulating an encystment reaction that is produced with the type of cells of the invaded tissue.
The thorn or the fungus can therefore cause, according to the case, an epithelioma, an adenocarcinoma, an osteosarcoma, a gliobastoma, and so on.

In the first moments of the invasion, the organism is able to send mature cells to contain the proliferating fungi: this is the phenomenon of a differentiated tumor. As the colonies become more powerful, and tissues are exhausted, cells become more and more immature up to anaplasia.
Furthermore, the ratio between differentiated tissues and connective tissue existing in an organ determines the reaction capability and thus the degree of malignancy of a neoplasia. The fewer noble cells there are, the more malignant and invasive the tumor becomes.

So, on the one hand we have noble tissue which cannot be attacked (muscles and nerves), and on the other the simple connective tissue. The glandular tissue which is halfway between these two elements, just because it is provided with that complex structure that confers to it a certain ability of encysting the fungi, can oppose their invasion by producing the phenomenon of the benign tumor. For example, if we consider the thyroid, we can see how in this gland neo-formations can take any graduation of malignancy even when they possess benign histological characteristics, as is the case for capsulated follicular carcinoma, long ago called metastasizing benign adenoma.

This can happen because the concept of a 'benign tumor' does not have an absolute value. In this case, even if it is true that fungin cells cannot normally go through the differentiated cells barrier, that does not mean that under particular conditions they cannot be successful.
It is for this reason that such neo-formations are considered 'odd' in oncology. But such oddities can be easily explained with the interpretation key of fungin infection. When the glandular tissue is exhausted, the benign tumor becomes a malignant one.

For all intents and purposes, it is always the same Candida attacking different tissues, each time adapting itself to the type of environment it finds. The specifications usually assigned to the various candidas (Candida Albicans, Krusei, Parapsilosis, Glabrata, Tropicalis and others) underestimate the fact that they all come from one single progenitor which, when it genetically mutates to attack the host, transforms itself into this or that stock.59

R.L. Hopfer for example found no less than four different Candida species in the post-mortem cultures of a leukemia patient.
N. Aksoycan demonstrated that seven different stocks of Candida actually have the same antigenic structure.
F.C. Odds reports how the same Candida stock can colonize different anatomical areas at different times.
J. Hellstein has found the common clonal origin in Candida Albicans for both commensal and pathogenic stocks.

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